Researchers have reported the nature of these genes and their role in early protein sowing in related proteins and plant physiology journals.
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According to the team, seed germination is a complex process that involves many biochemical, physiological, and size changes in seed seeds in response to appropriate environmental conditions, including humidity, temperature, air, and light. One of the first stages of germination in many plants is the first embryonic leaf known as the cotyledons. Cotyledons protect the soft shoots that will develop in the aerial parts of the plant and the timing of its opening is crucial.
“Adverse conditions such as unseasonal rainfall can lead to premature opening of the cotyledon, which can lead to seed damage and failure to maintain plant growth.
“It is important to be able to control the opening of the cotyledon to prevent unnecessary damage and death of the plant. That said, Interpol’s molecular control between the opening, light and BR is not yet well understood.
IISER scientists have shown that a protein encoded with a gene called BBX32 negatively controls light signaling and propagates BR signals to prevent the opening of cotyledons in model plant Arabidopsis belonging to the mustard family.
“This gene has been shown to integrate information from BR into external light conditions and internal signals to optimize the release of cotyledon. Researchers have shown that BBX32 is induced by BR and physically interacts with another protein called BJR1.
“Now that we have some information about the genes and proteins that control this process, we can modify the expression of these genes at the best time for seed emergence and establishment by inventing seed technology,” he said.
The team said such controls could potentially be used to protect seedbeds from harsh environmental conditions and to ensure higher agriculture.